Abstract
Mitochondria, the powerhouse of the cell, play a central role in cellular energy production, metabolism, and homeostasis. Emerging evidence suggests that mitochondrial dysfunction is a key factor in aging and the development of age-related diseases such as Alzheimer's disease, Parkinson's disease, and cardiovascular diseases. This article explores the mechanisms by which mitochondrial dysfunction contributes to aging, the role of mitochondrial DNA mutations, oxidative stress, and the potential therapeutic strategies aimed at improving mitochondrial function to delay aging and prevent age-related diseases.
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